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This acetaminophen impairs the balance between tromboxane and prostacyclin and causes thrombus formation .
Contrary to the inflammatory tissue, the endothelial cells possess a low level of peroxides, so they are not likely to inhibit paracetamol acetaminophen activity against COX-2 (14) .
It has been shown that oral administration of paracetamol at the dose of 500 mg decreases the amount of excreted with urine 2,3-dinor-6-keto PGF1α, the main stable inactive metabolite of prostacyclin, whose synthesis is mediated by endothelial acetaminophen COX-2 (62) .
Likewise, 50% reduction in this metabolite acetaminophen daily dosage limit acetaminophen overdose signs acetaminophen dosage pregnancy excretion in the urine of pregnant women was noted after ingestion of 1 g of paracetamol (63) .
Taking into consideration aforementioned results obtained by Hinz acetaminophen et al .
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